ipl-logo

Rest Membrane Potential Lab Report

698 Words3 Pages

Resting membrane potential Resting potential occurs when a neuron has –70mV charge at potassium equilibrium. Before we talk about potassium equilibrium we must first understand how the cell works. To start off Ions can not pass the bilayer of a cell they may only pass through the semipermeable membrane through Ion channels found in proteins with in the cell membrane. These Ion channels are semipermeable because they only allow certain Ions to pass through them. There are 2 different types of Ion channels there are gated and nongated channels. There are also two types of gated channels the first is a voltage gated channel will open depending on the charge of the Ion. The second type of gated channel is a neurotransmitter which opens when a chemical is attached to it. The Ion Channel that is used for …show more content…

Na+ and Cl- also contribute to reaching equilibrium. When the neuron is at equilibrium its charge is –70mV. If the cell gets a potassium leak it will use a sodium potassium pump in order to maintain the equilibrium. The way the pump works is buy using ATP to pump three sodium ions for every two potassium ions. Generation of action potential Action potential occurs when the neuron goes from equilibrium and depolarizes causing the neuron to become more positive. If the neuron depolarizes enough it will reach the threshold. Once it reaches the threshold the neuron will generate and action potential. Depolarization takes place at the axon hillock this location has a sodium voltage gated channel. This location of the neuron has the greatest amount of sodium. The permeability of sodium increases as the voltage increases. In order for depolarization to reach threshold it must reach –55mV creating an action potential which is an all or nothing

Show More
Related
1022 Words | 5 Pages

When the acetylcholine bonds with the protein receptors in the motor end plate, sodium and potassium gates in the region open at the same time

Read More
1789 Words | 8 Pages

An activity potential from a motor neuron triggers the arrival of Ca2+ particles from the sarcoplasmic

Read More
1047 Words | 5 Pages

So, the result was presented as the emergence of paralysis in the affected areas. Initial blockage of sodium ion channel totally deactivated an entire process in the neuromuscular junction. In other words, sodium ions mainly influence to the action potential generated in muscle cells, so TTX may inhibit muscle activity

Read More
413 Words | 2 Pages

Dayma Padron Explain how the structure of the plasma membrane influences the movement of oxygen (O2), carbon dioxide (CO2) and sodium ions (Na+). The cells looks like tiny and insecure organisms in our body, but the reality is that without its perfect cellular organization, and essential functions, our body will be fragile, and we would live a short life. The plasma membrane, which is the wall of the cells, are composed of half lipids and half proteins, and about 5 to 10 percent of carbohydrates (Cooper, 2000). The membrane form a phospholipid bilayer, with fluid texture (cytoplasm), and acts like a barrier to protect its components from the outside neighbors, receptor, control mobility, maintain shape, and ensure that every part perform its

Read More
606 Words | 3 Pages

Glucose enters the nerve terminal by passive transport. Glycolysis occurs in the neuronal cytoplasm and pyruvate molecule are generated. Pyruvate is transported into the mitochondria and an acetyl group derived from pyruvic acid combines with coenzyme-A present in the mitochondria to form acetyl coenzyme-A, which is transported back into the cytoplasm. Choline is actively transported into the neuronal terminal from the synaptic cleft via sodium and choline transporters. An enzyme choline acetyltransferase (ChAT) in the nerve terminals synthesizes acetylcholine from choline and acetyl coenzyme A.

Read More
679 Words | 3 Pages

For lower potassium concentrations, membrane potential was better predicted by Sodium reversal potential, and for higher Potassium concentration, Chloride reversal potential was a better predictor of the membrane potential. Rather than calculating different reversal potentials for

Read More
1151 Words | 5 Pages

Benzodiazepines open GABA-activated chloride channels, and allow chloride ions to enter the neuron. This makes the neuron negatively charged and resistant to excitation, or in other words, resistant to receiving an

Read More
1731 Words | 7 Pages

Explain the process by which light generates membrane potentials within the photoreceptor. The photoreceptors detect the light. The muscles of our body cause our eyes to move so that certain images are not in our retina’s view; the shape of the lens then changes. Synapses and bipolar cells are channels in which photoreceptors communicate, which then communicate through synapses with ganglion cells.

Read More
192 Words | 1 Pages

1. Chloride ions will diffuse into the cell, as it is moving from an area of high concentration, to an area of low concentration. Chloride ions will diffuse into the cell because the equilibrium potential of chloride ions is more negative than the membrane potential, therefore when chloride ions diffuse into the cell the equilibrium potential of chloride ions and the membrane potential will become more balance. If, by the process of active transport, chloride ions moved out of the cell this would create a bigger gap between the equilibrium potential of chloride ions and the membrane potential. 2.

Read More
440 Words | 2 Pages

Single channel conductance () and reversal potential (Vr) were respectively of 100 pS and of 0 mV for outward currents and of 30 pS and of 0 mV for outward currents. Furthermore, the channel activity (NPo) was voltage-dependent, with high open probability at negative potentials and a a voltage-dependent inactivation at positive potentials (Figure XC). These channel properties are in good agreement with previously published data (Nilius…). A second set of experiment was devoted to chacterize the channel biophysical properties in the absence of divalent ions in the pipette, with the purpose to attenuate calcium-dependent inactivation (Citazione), a prerequisite for long duration experiments. Figure XD shows a record of 30 s long of the patch currents from an oxaliplatin treated neuron in the presence of 1 M of icilin at Vm= 40 mV (upper trace) in which at least three channels were active.

Read More
2220 Words | 9 Pages

These were shown to display spikes of activity of EPSPs. The spikes in cell 2 match up with the sub-threshold activity in cell 1. This was expected. When cell 2 reaches an action potential with its amplitude at 12mV, in figure 4, has a knock on effect of slightly hyperpolarising the following spike in cell 1. This hyperpolarisation is due to the fact that cell 1 and cell 2 are linked and so a depolarisation in cell 2 will lead to a small hyperpolarisation in cell 1.

Read More
1566 Words | 7 Pages

The negatively charged protein molecules (A-) inside the neuron cannot cross the membrane. In integration to these selective ion channels, there is a pump that utilizes energy to move three sodium ions out of the neuron for every two potassium ions it inserts. Conclusively, when all these forces balance out, and the difference in the voltage between the inside and outside of the neuron is quantified, have the resting membrane potential. The resting membrane potential of a neuron is about -70 mV (mV=millivolt) - this designates that the inside of the neuron is 70 mV

Read More
895 Words | 4 Pages

Since equilibrium cannot be reached, an electrochemical driving force is generated which acts on the ions. It is derived by finding the difference between the membrane potential obtained and the equilibrium potential expected. The sign of the value of this force decides the direction of movement of ions. Since we have cations (positive ions), a positive value shows movement of ions outside the cell membrane and a negative value shows movement of ions inside the cell membrane. If the value is equal to that of the equilibrium potential, the driving force acting on the ion is 0.

Read More
414 Words | 2 Pages

After the ACh has found its way to the receptors and is bound to them, the muscle fibers become stimulated. The stimulated muscle fibers result in an impulse being generated. This impulse travels down the membrane until it reaches the Sarcoplasmic Reticulum. The Sarcoplasmic Reticulum is responsible for regulating the Ca2+ ion concentration.

Read More
1117 Words | 5 Pages

This movement against a concentration gradient is known as an active transport process which makes ATP the currency of the cell. • The stimulus received by the dendrites of a neuron causes the Na+ channels to open. An influx of N+ ions via diffusion starts to drive the potential to be positive. Even though neurons are sensitive to external stimuli, certain stimuli might not cause the potential to rise to the threshold level. This will tend to make the neuron revert to equilibrium.

Read More

More about Rest Membrane Potential Lab Report

Open Document