Myocardial infarction Essays

“Incidence of Posttraumatic Stress Disorder (PTSD) After Myocardial Infarction (MI) and predictors of PTSD Symptoms Post MI- A Brief Report” by Leila P. Rocha, Mary E. Charlson, Janey C. Peterson, Nimali Jayasinghe, Barnett Meyers, Martha L. Bruce, Carla Boutin-Foster. This journal article was published in the International Journal of Psychiatry in Medicine. The journal published the article in 2008 (Leila P. Rocha, et al., 2008). Myocardial Infarction (MI) is the name that is used by doctors and members

INTRODUCTION As the incidence of myocardial infarction is increased in modern era . Conduction defects are one of the most common complications which occur following acute myocardial infarction (AMI), which results in Increased mortality in these patients with Acute Myocardial infarction 1. conduction defects occur during Acute Myocardial infarction , have varied presentation. Atrio ventricular (AV) blocks associated with inferior wall infarction and bundle branch blocks are more commonly

Myocardial Infraction Myocardial infraction (MI) or acute myocardial (AMI) well known as a heart attack, it happens when blood travels to the portion of the heart and this will end up causing the clotting of blood and the muscle of the heart suffers (Wood MA, Spores R, Natske LT, et al 1980). In explanation, the vsd evidently is caused when the walls of the ventricles fails to close appropriately through pregnancy and this will outcome in a high blood pressure owing to the is high volumes of blood

INTRODUCTION Chest pain is the most usual symptom at presentation in patients with acute myocardial infarction (AMI), even if it ishighly subjective, and the pain characteristics, severity and emotional consequences may vary widely.1, 2 An asymptomatic AMI is not necessarily less severe than a symptomatic event. Diabetic and elderly subjects are those who generally present with a lower severity of chest pain and at the same time represent the subgroups of AMI patients withworse outcome.3 Chest pain

Running head: NSTEMI 1 NSTEMI 5 Non-ST Elevated Myocardial Infarction Randie Rains Columbia Basin College Description of NSTEMI A non-ST elevated myocardial infarction, sometimes referred to as NSTEMI, is best described as a heart attack, damaging the innermost layer of the heart. A heart attack or myocardial infarction (MI) occurs when the blood flow to the heart muscle has been obstructed, limited or reduced due to plaque accumulation inside the coronary arteries. Plaque buildup

Annually mortality from coronary heart disease claims an estimated 1.8 million lives in Europe, despite having fallen over recent decades (1). In the United Kingdom around 110,000 men and 65,000 women have an acute myocardial infarction every year, equivalent to one every three minutes (2). In 1772 William Heberden, the original describer of ‘angina pectoris’, recounted a case of a patient who clinically improved by working in the woods for half an hour each day. However, mobility limitations were

ACUTE MYOCARDIAL INFARCTION An acute MI occurs when myocardial cells have reached the threshold of ischemia; which in turn cause the body’s myocardial cell repair mechanism to first become overwhelmed and then to completely fail. When this mechanism fails, myocardial tissue necrosis causing irreparable tissue/cell death occurs. The following are possible contributors to myocardial ischemia and ultimately myocardial infarction (tissue necrosis and cell death): • Increased myocardial metabolic demand

Angina, Myocardial Infarction, and Cardiac Arrest Comparison and Contrast Hannah Bunce Fayetteville Technical Community College Angina, Myocardial Infarction, and Cardiac Arrest Comparison and Contrast Three of the most commonly occurring cardiac related events: angina, myocardial infarction, and cardiac arrest, are commonly confused. However, there is a huge difference between each of them. It is crucial to be informed of each of these cardiovascular emergencies and to be able to

AH314-Pathophysiology Case Study 4 Myocardial Infarction K.R. is a 46-year-old man admitted to the emergency department with unremitting chest discomfort. The pain started while he was shoveling snow from his walkway. He had experienced chest discomfort with activity previously, but the pain had subsided with rest and he sought no medical help. This time the pain did not subside and became increasingly severe, radiating to his left arm and lower jaw. In the emergency department, an ECG and cardiac

heart attack. The dangers and results of this horrific disease affect the lives of people all around the globe but primarily here at home in America. I am going to discuss the effects this disease has on many Americans each and every day. A myocardial infarction or what’s mostly known as a heart attack happens when a heart muscle doesn’t receive enough blood flow to sustain muscle activity. This occurs when the blood vessel is damaged and platelets clot up the damage to prevent bleeding. Over a long

Cardiogenic shock is associated with inadequate tissue perfusion that can cause cardiac failure, usually causing acute myocardial infarctions .RG is a 68-year-old man that has been admitted to the ICU after undergoing coronary artery bypass grafting. He has been diagnosed with history of ischaemic heart disease with two previous myocardial infarctions, hypertension and hypercholesterolaemia which he has been on maintenance therapy. He has been administered with dopamine at 3 mcg/kg/minute and titrated

that will make vitamin D has a positive influence on heart function. However, long term vitamin D deficiency can rise the production of PTH. This over production of PTH can cause valvular calcification, cardiac arrhythmia, arterial hypertension, myocardial calcification, and left ventricle hypertrophy. Vitamin D deficiency also can increase the rate of renin-angiotensin system. It has an important function in controlling blood pressure, electrolyte balance, and intravascular volume. However, vitamin

take the medication as needed for chest pain. He took one pill an hour ago and a second pill 10 minutes prior to calling 911. Prior to this evening’s chest pain, he ate at a buffet and consumed 4 whiskey drinks. His ECG is not indicative of a myocardial infarction. What are the clues that helped the paramedic determine why the nitroglycerin did not alleviate the man’s angina? The paramedics knew the chest pain was coming from heartburn. You get heartburn from overeating and drinking. Heartburn can

INTRODUCTION: Although perioperative hypotension is a common problem, its true incidence is largely unknown.[1] There is evidence that postoperative outcome, including the incidence of myocardial adverse events, may be linked to the prolonged episodes of perioperative hypotension. Despite this, there are very few comprehensive resources available in the literature regarding diagnosis and management of these not so uncommon clinical occurrences, especially during non-cardiac surgery.[1] Perioperative

This class of medications are important because they the most commonly used medications for cardiovascular diseases. Some of the common diseases that beta blockers treat are angina, heart failure, high blood pressure, atrial fibrillation, and myocardial

A bundle branch block is when the electrical impulses that are sent to the heart are delayed. A ventricle within the heart will contract slower than the other ventricle. This will make it harder for the heart to pump blood efficiently through the body. Bundle branch block is specifically diagnosed by its location and severity. Furthermore, doctors will call it right or left bundle branch block, or even first, second, and third depress atrioventricular node blocks. A bundle branch block anatomy

INTRODUCTION Cardiovascular disease is a general term that describes the disease of the heart or the blood vessels. These diseases can include many categories, some of which includes, angina, abnormal blood pressure for the age group, myocardial infarction, cardiac arrest, abnormal pulse, hypertensive emergency and others which will be discussed later on. The cardiovascular system is actually made up of a few parts, including the heart, blood and all the blood vessels (arteries, veins and capillaries)

Coronarythrombosis: a resultratherthanacauseof infarction?It wasassertedthat thefrequencyofcoronary thrombosis wasgreaterin patients who died late after the onset of symptoms than in those who died early. This observation formed the basis for the view that the thrombus was aresult ratherthan acause oftheinfarction

• Journal ‘The New England journal of Medicine’ 10th September 2009 volume -361 no.11 • Title - Ticagrelor versus Clopidogrel in Patients with Acute Coronary Syndromes • Authors – Lars Wallentin, M.D., Ph.D., Richard C. Becker, M.D., Andrzej Budaj, M.D., Ph.D., Christopher P. Cannon, M.D.,Hakan Emanuelsson, M.D., Ph.D., Claes Held, M.D., Ph.D., Jay Horrow, M.D., Steen Husted, M.D., D.Sc.,Stefan James, M.D., Ph.D., Hugo Katus, M.D., Kenneth W. Mahaffey, M.D., Benjamin M. Scirica, M.D., M.P.H.

Pseudoaneurysm A pseudoaneurysm happens when an artery is injured and blood leaks out to form a sac-like bulge. The bulge can break open, causing bleeding in the nearby tissues. CAUSES The most common cause of this condition is a procedure such as an angiogram in which a thin tube (catheter) is inserted into an artery. After an angiogram, the insertion site on the artery should close back up all the way. If it does not, blood may leak out of the artery. Other causes of a pseudoaneurysm include: