Case Study Of Brain's Quality Of Life
Mr. Alewine has been informed being untruthful about wanting to manipulate the system is wrong and resources are shelters in the community to aid with with his stressors. He expresses knowledge of knowing what he has done. Mr. Alewine does not appear to be exhibiting signs of agitation, aggression, or responding to internal
He denies any perceptive abnormalities are delusional thought content, but he does demonstrate some impaired reality testing. Psychiatric Hx: Kwalon was under the care of CSB from 2010-2011 where he was already provided the diagnosis of ADHD and mood disorder with conduct problems, for which he has been prescribed a combination of Abilify and Concerta. He has a history of assaultive, aggressive and disorderly behaviors. These medications were at that time continued with sporadic compliance. After several no show appointments in 2011 his medication management was transferred back over to his PCP.
Mr. Chapman appears apprehensive and his agitation amplifies. Mr. Chapman express that he has already told the nurse and others what he needs multiple times and request that I review his chart, which stated that he has lower back and chronic pain. He stated
n the article Secrets of the Brain, author Carl Zimmer explores the current research that is going into how the brain works. With all the ways that people are suffering due to brain related injuries or diseases there is a lot of incentive to understand how the brain works so that they can understand what is causing things like autism and dementia and how to cure paralysis. The intricacies of the brain are so vast that research has barely scratched the surface of learning about how it works let alone how to use the information to cure brain damage. “We can’t tell a schizophrenic brain from an autistic brain from a normal brain,” says Christof Koch. But the research that’s going on now, he believes, is moving neuroscience to a remarkable new
Does rTMS Normalizes Hypoactivity in Prefrontal Cortex? An fNIRS-based Randomized Sham – controlled Trial of rTMS Stimulation on the Right Dorsolateral Prefrontal Cortex for the Treatment of Panic Disorder Although panic disorder has been the most investigated anxiety disorder during the past 25 years (Roy-Byrne et al., 2006) treatment resistance issue in panic disorder still remains unsettled. . Cowley and Roy-Byrne (1997) who were investigating treatment failure in pharmacotherapy suggest that the main reason of unsuccessful treatment with medications is intolerable side effects of medication.
SUBJECTIVE INFORMATION: Wirach is a 61 year old male that suffers from migraine headaches that is focused in the occipital area. Today he was seen by myself Kareen Wong (Pharmacy student) and Anna Rogers (Clinical Pharmacist) for a medication review. When asked about his current list of medications, he is currently taking propranolol for heart palpitations, venlafaxine for depression, lorazepam for sleep, ranitidine for heart burn and senokot for constipation. In addition, he is on amlodipine, fosinopril, and hydrochlorothiazide for hypertension, and atorvastatin for dyslipidemia. For the past nine to ten years, he has been taking lorazepam 3 mg PO at bedtime to help with sleep.
An important part in the brain involved in memory, decision-making, and emotional responses is called the medial amygdala (MePD). The MePD is located within the temporal lobes of the brain and plays a major role in the life of complex vertebrates. MePD receives information pertaining to reproductive behavior from the hypothalamic portion of the brain (Johnson et al., 2008). The MePD is numerous in neurons and astrocytes. Astrocytes are neuroglial cells with many branches, or processes, that originate in the central nervous system.
Jay Thompson is a 35 year old male who visited his psychologist. He kept complaining that he doesn’t get enough sleep at night . He also stated that he just felt depressed lately and just had a hard time getting things right at work. He had told the psychologist, Dr.Monroe, that he has difficulty sleeping, cognition, and think he has amnesia or dementia because he always forgets things. Jay explained that the other doctors he visited had given him medications to take, but he still had hard time sleeping.
The patient is a 56 year old woman who is brought to the emergency room by her family because of change in mental status. This patient has had previous admissions for similar episodes with change in mental status. She has a past history of a hypoglycemic coma, as well as the sequelae of dementia and the old chart relates that she has had dramatic brain injury with severe depression. She has been on Paxil in the past. EEG done recently in this past May shows normal wave sounds with any proximal features but she does have significant sequelae as her baseline although findings in the past noted her to be only oriented x1.
Fear is a primitive human emotion vital for responding to physical and emotional danger. Although fear can be learned, in our evolutionary past, fear was essential as it enabled humans to respond to danger using the fight-or-flight response via the Sympathomedullary Pathway. An important neuroanatomical structure involved in the process of fear is the amygdala (LeDoux, 2003) which is part of the limbic system. Research has shown that the amygdala is implication in recognition of fear (Adolphs et al. 1995) and feelings of fear (e.g. Ketter et al. 1996). Evidence comes from a human case study (SM) by Damasio et al (1994) who used MRI scanning on a patient with Urbach-Wiethe disease which lead total atrophy of her bilateral amygdala.
Depression commonly affects a vast amount of patients in the primary care setting (L. Foldager, 2011). The increased cases of depression over the years are worrying (Mitchell et al., 2013). There is thus a need to enhance diagnosis and treatment. The purpose of the paper is to review an evidence-based clinical practice guideline (CPG) used to effectively diagnose and treat individual suspected of having depression by health practitioners in the primary care setting.
How does polypharmacy come about? •Unfortunately, as we all age our bodies start to grow old with us. Arthritis kicks in, consitpation, diarrhea, our hearts get worn, our kidneys get tired, we forget important things.
I feel that if a person where to have parts of their brains removed and replaced with implants that simulated the same functions that the part of the brain that was removed does, that the person still has what would be considered a “mind”. A person with implants that replace someones, Memory, thinking, emotional, reaction or other similar centers of the brain with a chip that behaved exactly as the part of the brain that was replaced did and had the same information in it would not only have a self and identity but have the same self and identity as before. according to the factionalism theory the mind is the brain all things that the mind is and is expressing as all contained in the brain. The mind/brain behaves the way it does because
Imagine going to school and really succeeding; you understand everything, you’re getting good grades and all the praise you can dream of from your parents and teachers. But then you move up and things get harder, you don’t understand everything, your grades are dropping and you are scared that you will no longer get that praise. You have two options, you can either take on the challenge and get back to where you used to be, or you can sit down when you feel threated by the hard work. In “Brainology” by author Carol S. Dweck, we are shown research concerning those two options or “mindsets” and how we can change them.
1. INTRODUCTION 1.1 Brain Asymmetry In general, any biological system shows some degree of asymmetry in their organization. From the highly intelligent system such as human to the lower animals, normal variation and specialization result in formation of asymmetries in both structure as well as function. In some mammalian system including human, the two brain hemisphere differ in their anatomy and function.
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