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Essay On Epilepsy

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The word “epilepsy” comes from the Greek and means "to be taken, seized or attacked" and refers to chronic seizures. Epilepsy, the term is used for recurrent seizures, is one of the most common neurological disorders of childhood. The International League Against Epilepsy (ILAE) and the International Bureau for Epilepsy (IBE) define epilepsy as a disorder of the brain characterized by an enduring predisposition to generate epileptic seizures and by the biologic, cognitive, psychological, and social consequences of this condition (Pillitteri, 2010; Algreeshah et al., 2013 and Barza, 2014).

It is a life-long tendency, though the seizures may start at any time during life and occur sporadically or frequently. Some of the epilepsies are confined …show more content…

These discharges may be focal or diffuse (Tejada et al., 2014).

Seizures are further characterized as epileptic and non-epileptic. Epileptic seizures are those associated with corresponding electrographic activity in the brain. Non epileptic seizures, conversely, are clinical events that do not correspond to electrographic activity. They can be a response to physiologic event, such as fever, toxins, or hypoxia, or a reaction to some type of psychic stressor (psychogenic seizures) (Dimaro et al., 2014 and Hockenberry & Wilson, 2015).

An epileptic seizure is a sudden and transient occurrence of signs and/or symptoms that are the result of an abnormal activity of the brain. Epileptic seizures are the common and defining component of the disorder that is referred to as epilepsy. In other words, epilepsy is the tendency to have repeated (two or more) spontaneous epileptic seizures (Roswall, 2010; Mustapha et al., 2013 and Fisher et al.,

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People of all ages can get it and a significant sign of diagnosing the condition is experiencing two or more seizures in a 24 hour period (“The Epilepsies” 2016). Although there is no cure for epilepsy, the condition can be controlled with anticonvulsants drugs. I personally have prior knowledge to the condition from my AP Biology class. I immediately thought of my senior year of high school, when I first was taught about epilepsy. My friend Nadine showed us a documentary which showed a old man who would suddenly fall out of consicouness during his daily tasks; I remember this documentary very well because it was a scary sight for me

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Social influences were also a big factor in my episode of seizing. My best friend went to find help, the teachers helped carry me into a different room, and everyone that was near stopped what they were doing to crowd over me. Once I was in the different room an upperclassman, which had been diagnosed with epilepsy, explained that after she seizes she would always throw up too. She told me that it was all going to be fine, and that really helped me. Developmental influences also have occurred.

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Moreover, typical absences usually last 9–12 s (Hooge, & De Deyn, 2001). Indeed, mutations of genes coding for GABA A receptors and T-type calcium channels have been linked to AS. More recent studies have found that Glucose transporters might also be linked to AS, however data is limited in this respect and offers further room for exploration. The pathophysiological theories hypothesized to date, have clearly always recognised the contribution of two forebrain structures, the thalamus and the cerebral cortex, and their fundamental roles in the generation of seizures.

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